<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0034-7744</journal-id>
<journal-title><![CDATA[Revista de Biología Tropical]]></journal-title>
<abbrev-journal-title><![CDATA[Rev. biol. trop]]></abbrev-journal-title>
<issn>0034-7744</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Costa Rica]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0034-77442025000100030</article-id>
<article-id pub-id-type="doi">10.15517/rev.biol.trop..v73i1.58890</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Determinación molecular de Helicobacter pylori y detección de beta-lactamasas en aislamientos de pacientes de Antioquia-Colombia]]></article-title>
<article-title xml:lang="en"><![CDATA[Molecular determination of Helicobacter pylori and detection of beta-lactamases in clinical isolates from patients in Antioquia-Colombia]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Arango]]></surname>
<given-names><![CDATA[Yesenia]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Mesa]]></surname>
<given-names><![CDATA[Laura]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Atehortúa]]></surname>
<given-names><![CDATA[José]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Escobar]]></surname>
<given-names><![CDATA[Rene]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández]]></surname>
<given-names><![CDATA[Sergio]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Castro]]></surname>
<given-names><![CDATA[Andrés]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Bautista]]></surname>
<given-names><![CDATA[Henry]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Vélez-Gómez]]></surname>
<given-names><![CDATA[Diego]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Martínez]]></surname>
<given-names><![CDATA[Alonso]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Salazar]]></surname>
<given-names><![CDATA[Beatriz]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pérez-Cala]]></surname>
<given-names><![CDATA[Tania]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad de Antioquia Facultad de Medicina Grupo Bacterias &amp; Cáncer, Departamento de Microbiología y Parasitología]]></institution>
<addr-line><![CDATA[Medellín Antioquia]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad de Antioquia Escuela de Microbiología ]]></institution>
<addr-line><![CDATA[Medellín Antioquia]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Hospital Universitario San Vicente Fundación Unidad Funcional en Enfermedades Digestivas y Trasplantes ]]></institution>
<addr-line><![CDATA[Medellín Antioquia]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Gastroenterología Clínica-quirúrgica Unidad Gastroenterológica SAS ]]></institution>
<addr-line><![CDATA[Medellín Antioquia]]></addr-line>
<country>Colombia</country>
</aff>
<aff id="Af5">
<institution><![CDATA[,Universidad de Santander Facultad de Ciencias de la Salud Grupo de Investigación en Manejo Clínico CliniUDES]]></institution>
<addr-line><![CDATA[Bucaramanga Santander]]></addr-line>
<country>Colombia</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<volume>73</volume>
<numero>1</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_arttext&amp;pid=S0034-77442025000100030&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_abstract&amp;pid=S0034-77442025000100030&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_pdf&amp;pid=S0034-77442025000100030&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Introducción: Helicobacter pylori presenta diferencias en la resistencia a antibiótica en América y Colombia; a pesar de tener varios esquemas de tratamiento que emplean la amoxicilina en el departamento de Antioquia-Colombia, así como en varios países del continente se desconoce el patrón de susceptibilidad genotípica y fenotípica para este antibiótico.  Objetivo:  Determinar la frecuencia de infección por H. pylori por amplificación del gen ureA y la susceptibilidad a amoxicilina mediante la detección de beta-lactamasas en aislamientos de H. pylori de pacientes con enfermedades gastroduodenales procedentes de Antioquia-Colombia.  Métodos:  Estudio analítico transversal con 179 pacientes que acudieron a las unidades de endoscopía de siete instituciones de salud. Se recolectó información de datos sociodemográficos y factores de riesgo. Además, se tomaron biopsias de antro y cuerpo para aislamiento microbiológico y detección molecular de H. pylori. Los datos sociodemográficos se evaluaron a partir de análisis de frecuencia y estadística bivariada. El ADN obtenido de las biopsias se amplificó por PCR para identificar el gen ureA, la producción de beta-lactamasas se evaluó con la prueba de nitrocefin y la amplificación de los genes TEM y SHV.  Resultados:  Del total de pacientes, 61 % (109 / 179) fueron positivos para H. pylori y de 84 aislamientos obtenidos por cultivo, ninguno fue positivo para la prueba de nitrocefin. En pacientes positivos para H. pylori se encontraron diferencias significativas en el estrato socioeconómico, régimen y ubicación geográfica. Dos pacientes fueron positivos en la determinación molecular de beta-lactamasas a partir de biopsias, aunque en ningún aislamiento se encontró los genes SHV o TEM.  Conclusión:  La frecuencia de H. pylori por detección del gen ureA es mayor comparada con estudios previos y presenta similitud con registros de otras regiones colombianas, por lo que sería una prueba diagnóstica de utilidad en el país. Finalmente, no se encontró aislamientos positivos para TEM y SHV.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Introduction: Helicobacter pylori exhibit variations in antibiotic resistance in America and Colombia. Despite the use of various treatment regimens containing amoxicillin in the department of Antioquia, Colombia, as well as in several countries across the continent, the pattern of genotypic and phenotypic susceptibility to this antibiotic remains unknown.  Objective:  To determine the frequency of H. pylori infection by detecting the ureA gene and amoxicillin susceptibility through beta-lactamases in H. pylori isolates from patients with gastroduodenal diseases in Antioquia-Colombia.  Methods:  Cross-sectional analytical study with 179 patients attending endoscopy units of seven healthcare institutions. Sociodemographic and risk factor data were collected. Moreover, biopsies from the antrum and body were taken for microbiological isolation and molecular detection of H. pylori. The sociodemographic data were assessed through frequency analysis and bivariate statistics. DNA obtained from the biopsies was amplified by PCR for the ureA gene, and beta-lactamase production was evaluated using the nitrocefin test as well as the amplification of the TEM and SHV genes.  Results:  It was found that 61 % (109 / 179) of patients tested positive for H. pylori; out of 84 isolates obtained by culture, none tested positive for the nitrocefin test. Significant differences were found in socioeconomic status, healthcare system, and geographical location in patients positive for the ureA gene. Two patients tested positive for molecular detection of beta-lactamases from biopsies; however, SHV and TEM genes were not found in any isolate.  Conclusions:  The frequency of H. pylori for ureA-gene detection is higher than previous studies and similar to other reports in other Colombian regions; consequently, this will become a good diagnostic test in the country. Finally, none of the H. pylori isolates tested positive for beta-lactamases TEM and SHV.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Helicobacter pylori]]></kwd>
<kwd lng="en"><![CDATA[microbial drug resistance]]></kwd>
<kwd lng="en"><![CDATA[stomach diseases]]></kwd>
<kwd lng="en"><![CDATA[beta-lactamases]]></kwd>
<kwd lng="en"><![CDATA[amoxicillin]]></kwd>
<kwd lng="en"><![CDATA[Colombia.]]></kwd>
<kwd lng="es"><![CDATA[Helicobacter pylori]]></kwd>
<kwd lng="es"><![CDATA[farmacorresistencia microbiana]]></kwd>
<kwd lng="es"><![CDATA[gastropatías]]></kwd>
<kwd lng="es"><![CDATA[beta-lactamasas]]></kwd>
<kwd lng="es"><![CDATA[amoxicilina]]></kwd>
<kwd lng="es"><![CDATA[Colombia.]]></kwd>
</kwd-group>
</article-meta>
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