<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>2215-3411</journal-id>
<journal-title><![CDATA[Odovtos International Journal of Dental Sciences]]></journal-title>
<abbrev-journal-title><![CDATA[Odovtos]]></abbrev-journal-title>
<issn>2215-3411</issn>
<publisher>
<publisher-name><![CDATA[Facultad de Odontología. Universidad de Costa Rica]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S2215-34112025000300111</article-id>
<article-id pub-id-type="doi">10.15517/m983tw41</article-id>
<title-group>
<article-title xml:lang="en"><![CDATA[The histone deacetylase inhibitor sodium butyrate stimulates adipogenesis through a limited transcriptional switch in periodontal ligament-derived stem cells]]></article-title>
<article-title xml:lang="es"><![CDATA[El inhibidor de la histona desacetilasa butirato de sodio estimula la adipogénesis a través de un cambio transcripcional limitado en células troncales derivadas de ligamento periodontal]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Torres-Nájera]]></surname>
<given-names><![CDATA[Anahí]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Serralta-Interían]]></surname>
<given-names><![CDATA[Angélica Anahí]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rivera-Solís]]></surname>
<given-names><![CDATA[Rodrigo Arturo]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Nic-Can]]></surname>
<given-names><![CDATA[Geovanny]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Carrillo-Cocom]]></surname>
<given-names><![CDATA[Leydi]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Rodas-Junco]]></surname>
<given-names><![CDATA[Beatriz Adriana]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
<xref ref-type="aff" rid="Aaf"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Universidad Autónoma de Yucatán Facultad de Ingeniería Química ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Autónoma de Yucatán Facultad de Odontología Laboratorio de Células Troncales-]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Mexico</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Autónoma de Yucatán Facultad de Ingeniería Química SECIHTI]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Mexico</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2025</year>
</pub-date>
<volume>27</volume>
<numero>3</numero>
<fpage>111</fpage>
<lpage>128</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_arttext&amp;pid=S2215-34112025000300111&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_abstract&amp;pid=S2215-34112025000300111&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_pdf&amp;pid=S2215-34112025000300111&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Adipogenic differentiation plays a crucial role in adipose tissue biology, an endocrine organ that regulates energy storage and hormone secretion. Dysfunction in this process contributes to metabolic diseases such as obesity and type II diabetes. In vitro models have been developed to investigate the mechanisms of adipogenesis, with periodontal ligament stem cells (PDLSCs) emerging as a promising model due to their multipotent capacity. Previous studies have shown that epigenetic manipulation can enhance the adipogenic response in various cell lines. Acetylation of lysine 9 on histone H3 (H3K9ac) is associated with the activation of key genes, such as PPAR&#947;-2. In this study, we evaluated whether class I histone deacetylase inhibitors, such as valproic acid (VPA) and sodium butyrate (NaBu), both short-chain fatty acids, can increase H3K9 acetylation and influence adipogenic differentiation. We used 1, 4, and 8 mM VPA concentrations and 1, 2, and 5 mM NaBu to assess their effects on cell viability, morphology, H3K9ac distribution, and adipogenic differentiation. The results indicated that cells treated with 5 mM NaBu exhibited morphological changes, reduced viability, increased H3K9ac signal intensity, and enhanced intracellular lipid deposition. These results infer that inhibition of HDACs by NaBu increases plasticity toward adipogenesis of PDLSCs through a limited transcriptional change in their key genes.]]></p></abstract>
<abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen La diferenciación adipogénica desempeña un papel crucial en la biología del tejido adiposo, un órgano endocrino que regula el almacenamiento de energía y la secreción hormonal. La disfunción en este proceso contribuye a enfermedades metabólicas como la obesidad y la diabetes tipo II. Se han desarrollado modelos in vitro para investigar los mecanismos de la adipogénesis, y las células troncales de ligamento periodontal (CTLP) emergen como un modelo prometedor debido a su capacidad multipotente. Estudios previos han demostrado que la manipulación epigenética puede mejorar la respuesta adipogénica en varias líneas celulares. La acetilación de la lisina 9 en la histona H3 (H3K9ac) se asocia con la activación de genes clave, como PPAR&#947;-2. En este estudio, evaluamos si los inhibidores de la histona desacetilasa de clase I, como el ácido valproico (VPA) y el butirato de sodio (NaBu), ambos ácidos grasos de cadena corta, pueden aumentar la acetilación de H3K9 e influir en la diferenciación adipogénica. Utilizamos concentraciones de VPA de 1, 4 y 8 mM y NaBu de 1, 2 y 5 mM para evaluar sus efectos sobre la viabilidad celular, la morfología, la distribución de H3K9ac y la diferenciación adipogénica. Los resultados indicaron que las células tratadas con NaBu de 5 mM presentaron cambios morfológicos, menor viabilidad, mayor intensidad de la señal de H3K9ac y mayor deposición lipídica intracelular. Estos resultados sugieren que la inhibición de las HDAC por NaBu aumenta la plasticidad hacia la adipogénesis de las CTLP mediante un cambio transcripcional limitado en sus genes clave.]]></p></abstract>
<kwd-group>
<kwd lng="en"><![CDATA[Periodontal ligament stem cells]]></kwd>
<kwd lng="en"><![CDATA[Histone acetylation]]></kwd>
<kwd lng="en"><![CDATA[Adipogenesis]]></kwd>
<kwd lng="en"><![CDATA[Histone deacetylase inhibitors]]></kwd>
<kwd lng="es"><![CDATA[Células troncales de ligamento periodontal]]></kwd>
<kwd lng="es"><![CDATA[Acetilación de histonas]]></kwd>
<kwd lng="es"><![CDATA[Adipogénesis]]></kwd>
<kwd lng="es"><![CDATA[Inhibidores de histonas desacetilasas]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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