<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1659-1321</journal-id>
<journal-title><![CDATA[Agronomía Mesoamericana]]></journal-title>
<abbrev-journal-title><![CDATA[Agron. Mesoam]]></abbrev-journal-title>
<issn>1659-1321</issn>
<publisher>
<publisher-name><![CDATA[Universidad de Costa Rica]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1659-13212026000100004</article-id>
<article-id pub-id-type="doi">10.15517/jdr4y941</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Genes de resistencia a Diaporthe phaseolorum var. caulivora en Glycine max: enfoques cualitativos y cuantitativos]]></article-title>
<article-title xml:lang="en"><![CDATA[Resistance genes to Diaporthe phaseolorum var. caulivora in Glycine max: qualitative and quantitative approaches]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Cuba Amarilla]]></surname>
<given-names><![CDATA[Mario Manuel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Peruzzo]]></surname>
<given-names><![CDATA[Alejandra María]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pioli]]></surname>
<given-names><![CDATA[Rosanna Nora]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Hernández]]></surname>
<given-names><![CDATA[Facundo Ezequiel]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Balaban]]></surname>
<given-names><![CDATA[David Mario]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Pratta]]></surname>
<given-names><![CDATA[Guillermo Raúl]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto de Biotecnología Agrícola  ]]></institution>
<addr-line><![CDATA[ Asunción]]></addr-line>
<country>Paraguay</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Universidad Nacional de Rosario Facultad de Ciencias Agrarias Cátedra de Fitopatología]]></institution>
<addr-line><![CDATA[ Zavalla]]></addr-line>
<country>Argentina</country>
</aff>
<aff id="Af3">
<institution><![CDATA[,Universidad Nacional de Rosario Facultad de Ciencias Agrarias Cátedra de Fitopatología]]></institution>
<addr-line><![CDATA[ Zavalla]]></addr-line>
<country>Argentina</country>
</aff>
<aff id="Af4">
<institution><![CDATA[,Universidad Nacional de Rosario Facultad de Ciencias Agrarias Consejo Nacional de Investigaciones Científicas y Técnicas]]></institution>
<addr-line><![CDATA[ Rosario]]></addr-line>
<country>Argentina</country>
</aff>
<aff id="Af5">
<institution><![CDATA[,Universidad Nacional de Rosario Facultad de Ciencias Agrarias Consejo Nacional de Investigaciones Científicas y Técnicas]]></institution>
<addr-line><![CDATA[ Zavalla]]></addr-line>
<country>Argentina</country>
</aff>
<aff id="Af6">
<institution><![CDATA[,Universidad Nacional de Rosario Facultad de Ciencias Agrarias Cátedra de Genética]]></institution>
<addr-line><![CDATA[ Zavalla]]></addr-line>
<country>Argentina</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2026</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2026</year>
</pub-date>
<volume>37</volume>
<numero>1</numero>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_arttext&amp;pid=S1659-13212026000100004&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_abstract&amp;pid=S1659-13212026000100004&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_pdf&amp;pid=S1659-13212026000100004&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Introducción.  El conocimiento del modo de herencia de la respuesta al cancro del tallo de la soja (CTS) causado por el hongo Diaporthe phaseolorum var. caulivora (Dpc) es necesario para incorporar resistencia genética al cultivo. Objetivo. Aplicar enfoques de la genética cualitativa y cuantitativa clásicas para la identificación del gen Rdc1 de resistencia a la cancrosis del tallo de soja causada por Dpc. Materiales y métodos. El estudio se realizó en la Universidad Nacional de Rosario, Argentina, entre los años 2012 y 2020. Se evaluaron las poblaciones segregantes PS-258, PS-157 y PS-172, inoculadas con la cepa Dpc16 en generaciones tempranas (familias F2:3) y avanzadas (familias F3:5 y F4), bajo un diseño experimental completamente aleatorizado. Resultados. El enfoque cualitativo detectó inequivocamente un locus mendeliano en la población PS-258. Mediante el enfoque cuantitativo, la heredabilidad estimada para PS-258 fue de 0,68 (incidencia-F2:3), 0,17 (incidencia-F3:5), 0,55 (severidad-F2:3) y 0,18 (severidad-F3:5), con un número de genes estimado de 0,78 (redondeado a uno, solo para severidad). En PS-157, la heredabilidad fue de 0,76 (incidencia-F2:3), 0,48 (incidencia- F4), 0,60 (severidad-F2:3) y 0,21 (severidad- F4), y el número de genes se estimó en 4,1 para incidencia y 0,18 para severidad, lo que indica que ambos progenitores aportaron alelos de resistencia en diferentes loci. En el cruzamiento PS-172, la heredabilidad fue de 0,89 (incidencia-F2:3), 0,92 (incidencia- F4), 0,04 (severidad-F2:3) y 0,13 (severidad- F4), con un número de genes de 0,2 para incidencia y 0,24 para severidad, consistente con la ausencia de genes Rdc conocidos en los progenitores. En todas las poblaciones se observaron altas correlaciones fenotípicas entre incidencia y severidad. Conclusión. La aplicación combinada de enfoques cualitativos y cuantitativos permitió confirmar la presencia del gen mendeliano Rdc1 en el cruzamiento PS-258, mientras que en PS-157 y PS-172 el enfoque cuantitativo evidenció la participación de más de un gen mayor y efectos poligénicos.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract  Introduction.  Understanding the inheritance pattern of the response to soybean stem canker (SSC), caused by Diaporthe phaseolorum var. caulivora (Dpc), is essential for the incorporation of genetic resistance into soybean breeding programs. Objective. To apply classical qualitative and quantitative genetic approaches to identify the presence of the Rdc1 gene conferring resistance to soybean stem canker caused by Dpc. Materials and methods. The study was conducted at the Universidad Nacional del Rosario, Argentina, between 2012 and 2020. Segregating populations PS-258, PS-157, and PS-172 were evaluated after inoculation with the Dpc16 strain in early (F2:3 families) and advanced generations (F3:5 and F4 families). A completely randomized experimental design was used. Results. The qualitative approach unequivocally identified a single Mendelian locus only in population PS-258. Using the quantitative approach, heritability estimates for PS-258 were 0.68 (F2:3 incidence), 0.17 (F3:5 incidence), 0.55 (F2:3 severity), and 0.18 (F3:5 severity). The estimated number of genes was 0.78 (rounded to one, for severity only). In PS-157, heritability values were 0.76 (F2:3 incidence), 0.48 (F4 incidence), 0.60 (F2:3 severity), and 0.21 (F4 severity), with an estimated gene number of 4.1 for incidence and 0.18 for severity, indicating that both parents contribute resistance alleles at different loci. For PS-172, heritability estimates were 0.89 (F2:3 incidence), 0.92 (F4 incidence), 0.04 (F2:3 severity), and 0.13 (F4 severity), and the estimated number of genes was 0.2 for incidence and 0.24 for severity, consistent with the absence of known Rdc genes in the parental lines. In all populations, phenotypic correlations between incidence and severity were high. Conclusion. The combined use of qualitative and quantitative genetic approaches confirmed the presence of the Mendelian resistance gene Rdc1 in the PS-258 cross, whereas in PS-157 and PS-172, the quantitative approach revealed the involvement of more than one major gene and polygenic effects.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[genética cualitativa]]></kwd>
<kwd lng="es"><![CDATA[genética cuantitativa]]></kwd>
<kwd lng="es"><![CDATA[resistencia a enfermedades]]></kwd>
<kwd lng="es"><![CDATA[biometría]]></kwd>
<kwd lng="es"><![CDATA[correlación]]></kwd>
<kwd lng="en"><![CDATA[qualitative genetics]]></kwd>
<kwd lng="en"><![CDATA[quantitative genetics]]></kwd>
<kwd lng="en"><![CDATA[disease resistance]]></kwd>
<kwd lng="en"><![CDATA[biometrics]]></kwd>
<kwd lng="en"><![CDATA[correlation]]></kwd>
</kwd-group>
</article-meta>
</front><back>
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