<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>1409-0015</journal-id>
<journal-title><![CDATA[Medicina Legal de Costa Rica]]></journal-title>
<abbrev-journal-title><![CDATA[Med. leg. Costa Rica]]></abbrev-journal-title>
<issn>1409-0015</issn>
<publisher>
<publisher-name><![CDATA[Asociación Costarricense de Medicina Forense]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S1409-00152017000200090</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[El endotelio: estructura, función y disfunción endotelial]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Carvajal Carvajal]]></surname>
<given-names><![CDATA[Carlos]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Hospital de Guápiles Laboratorio Clínico ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>12</month>
<year>2017</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>12</month>
<year>2017</year>
</pub-date>
<volume>34</volume>
<numero>2</numero>
<fpage>90</fpage>
<lpage>100</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_arttext&amp;pid=S1409-00152017000200090&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_abstract&amp;pid=S1409-00152017000200090&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_pdf&amp;pid=S1409-00152017000200090&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen El endotelio se puede definir como una monocapa que separa los tejidos de la sangre. Las células endoteliales tienen una multitud de funciones que son específicas a su localización. La principal función del endotelio es regular el flujo y la perfusión sistémicos a través de cambios en el diámetro y en el tono vascular. La disfunción endotelial (DE) comprende una pérdida del balance entre los factores vasodilatadores y vasoconstrictores derivados del endotelio, donde el estado vasoconstrictor llega a ser dominante, llevando a cambios patofisiológicos progresivos. Colectivamente, estos cambios endoteliales exhiben características proinflamatorias, prooxidantes, proliferativas, procoagulantes y de adhesión vascular. El proceso de aterosclerosis comienza temprano en la vida y la DE contribuye a la aterogénesis en cada fase de la aterosclerosis. La disfunción endotelial es un marcador de la aterosclerosis y es el precursor más claramente definido de la aterosclerosis. El estrés oxidativo es el mecanismo celular subyacente para el desarrollo de la DE. La inflamación es otro mecanismo subyacente a la DE. Hay una relación causal entre el estrés oxidativo y la inflamación. El estrés oxidativo puede amplificar las vías de señalización vasculares inflamatorias y la inflamación incrementa el estrés oxidativo. La función endotelial puede ser medida mediante su capacidad para ejecutar varias funciones, incluyendo el tono vasomotor, la expresión de moléculas de adhesión y el mantenimiento de un microambiente antitrombótico. En la investigación clínica la función endotelial se detecta midiendo los cambios en el tono vasomotor en respuesta a varios estímulos. Un número de biomarcadores se han identificado y utilizado como indicadores de función endotelial.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract The endothelium could be defined as a cellular monolayer separating all tissues from the bloodflow. Endothelial cells have a myriad of functions that are specific to their location. The main function of endothelium is to regulate systemic blood flow and tissue perfusión through changes in vessel diameter and tone vascular. Endothelial dysfunction (ED) comprises a loss of balance between endothelial-derived vasodilatory and vasoconstrictory factors, where the pro-vasoconstrictory state become dominant, leading to progressive pathophysiological changes. Collectively, these endothelial changes exhibit proinflammatory, pro-oxidant, proliferative, procoagulation and pro-vascular adhesion features. The process of atherosclerosis begins early in life, and endothelial dysfunction contributes to atherogenesis at every phase of atherosclerosis. Endothelial dysfunction is a marker of atherosclerosis and it is the most clearly defined early precursor of atherosclerosis. Oxidative stress to be the common underlying cellular mechanisms for the development of ED. Inflammation is another common underlying mechanism of ED. There is a causal relationship between oxidative stress and inflammation. Oxidative stress may amplify vascular inflammation signalling pathways, and conversely inflammation increases oxidative stress. Endothelial function can be assessed by assaying its capacity to perform its various functions, including regulation of vasomotor tone, expression of adhesion molecules and maintenance of an anti-thrombotic microenvironment. In clinical Research, endothelial function is tipically assessed by measuring changes in vasomotor tone in response to various stimuli. A number of biomarkers have been identified and used as indicators of endothelial function.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Endotelio]]></kwd>
<kwd lng="es"><![CDATA[disfunción endotelial]]></kwd>
<kwd lng="es"><![CDATA[aterosclerosis]]></kwd>
<kwd lng="en"><![CDATA[Endothelium]]></kwd>
<kwd lng="en"><![CDATA[endothelial dysfunction]]></kwd>
<kwd lng="en"><![CDATA[atherosclerosis]]></kwd>
</kwd-group>
</article-meta>
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