<?xml version="1.0" encoding="ISO-8859-1"?><article xmlns:mml="http://www.w3.org/1998/Math/MathML" xmlns:xlink="http://www.w3.org/1999/xlink" xmlns:xsi="http://www.w3.org/2001/XMLSchema-instance">
<front>
<journal-meta>
<journal-id>0379-3982</journal-id>
<journal-title><![CDATA[Revista Tecnología en Marcha]]></journal-title>
<abbrev-journal-title><![CDATA[Tecnología en Marcha]]></abbrev-journal-title>
<issn>0379-3982</issn>
<publisher>
<publisher-name><![CDATA[Instituto Tecnológico de Costa Rica]]></publisher-name>
</publisher>
</journal-meta>
<article-meta>
<article-id>S0379-39822022000100044</article-id>
<article-id pub-id-type="doi">10.18845/tm.v35i1.5429</article-id>
<title-group>
<article-title xml:lang="es"><![CDATA[Caracterización de cultivos neuronales de hipocampo murino como modelo de estudio de la señalización NRGs/ErbB4]]></article-title>
<article-title xml:lang="en"><![CDATA[Murine hippocampal neuronal culture characterization as a model for the study of NRGs/ErbB4 signaling]]></article-title>
</title-group>
<contrib-group>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Monge-Mora]]></surname>
<given-names><![CDATA[Luis Felipe]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
<contrib contrib-type="author">
<name>
<surname><![CDATA[Soto-Bernardini]]></surname>
<given-names><![CDATA[María Clara]]></given-names>
</name>
<xref ref-type="aff" rid="Aff"/>
</contrib>
</contrib-group>
<aff id="Af1">
<institution><![CDATA[,Instituto Tecnológico de Costa Rica Escuela de Biología ]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Costa Rica</country>
</aff>
<aff id="Af2">
<institution><![CDATA[,Instituto Tecnológico de Costa Rica Centro de Investigación en Biotecnología (CIB) Escuela de Biología]]></institution>
<addr-line><![CDATA[ ]]></addr-line>
<country>Costa Rica</country>
</aff>
<pub-date pub-type="pub">
<day>00</day>
<month>03</month>
<year>2022</year>
</pub-date>
<pub-date pub-type="epub">
<day>00</day>
<month>03</month>
<year>2022</year>
</pub-date>
<volume>35</volume>
<numero>1</numero>
<fpage>44</fpage>
<lpage>52</lpage>
<copyright-statement/>
<copyright-year/>
<self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_arttext&amp;pid=S0379-39822022000100044&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_abstract&amp;pid=S0379-39822022000100044&amp;lng=en&amp;nrm=iso"></self-uri><self-uri xlink:href="http://www.scielo.sa.cr/scielo.php?script=sci_pdf&amp;pid=S0379-39822022000100044&amp;lng=en&amp;nrm=iso"></self-uri><abstract abstract-type="short" xml:lang="es"><p><![CDATA[Resumen  Las neuregulinas (NRGs) son una familia de factores de crecimiento tipo epidérmico que actúan modulando diversas respuestas celulares a través de la interacción con los receptores ErbBs. Las NRG1 y NRG2 son las principales en el Sistema Nervioso Central (SNC) y su principal receptor en el cerebro es el ErbB4. La señalización NRG1/ErbB4 ha sido involucrada en procesos relevantes para la formación y mantenimiento del SNC como la migración de interneuronas, la mielinización, la neurotransmisión, la modulación de sinapsis, entre otras. Por otro lado, la NRG2 también se une y activa al receptor ErbB4, sin embargo, su señalización ha sido menos explorada. Existe evidencia de que alteraciones funcionales en estos módulos de señalización podrían provocar disfunciones en redes neuronales, de relevancia en el contexto de enfermedades neuropsiquiátricas. Por lo tanto, es de gran importancia contar con modelos in vitro para el estudio de su señalización, caracterizados en cuanto a la proporción de tipos de neuronas en las cuales las proteínas se expresan, y a la expresión de sus genes. En el presente trabajo se determinó una proporción aproximada de 85% de neuronas glutamatérgicas y 15% de interneuronas GABAérgicas en cultivos primarios de neuronas de hipocampo de ratones C57BL/6 preparados del día embrionario (E) 18, similar a lo que se ha reportado in vivo e in vitro. Además, se determinó la expresión de los genes Nrg1 tipo III, Nrg2 y ErbB4 para el día in vitro (DIV) 7, siendo la expresión de Nrg1 tipo III mayor que Nrg2, similar a lo reportado en el hipocampo in vivo. Esto sugiere que los cultivos pueden ser utilizados como modelos in vitro válidos para el estudio de la señalización de las NRGs.]]></p></abstract>
<abstract abstract-type="short" xml:lang="en"><p><![CDATA[Abstract Neuregulins (NRGs) comprise a family of EGF-like growth factors that modulate diverse cellular responses by interacting with their receptors ErbBs. In the Central Nervous System (CNS) the principal members include NRG1 and NRG2, and their principal receptor is ErbB4. The NRG1/ ErbB4 signaling has been involved in relevant processes for the development and maintenance of the CNS such as interneuron migration, myelinization, neurotransmission, and synapse modulation. NRG2 can also bind to ErbB4 and activate it: however, the NRG2/ErbB4 signaling remains less studied. There is evidence that functional disturbances in these signaling modules could lead to neuronal networks dysfunction, of importance in the context of neuropsychiatric diseases. Therefore, a characterized in vitro model in terms of the neuronal population in which these proteins are expressed, and their gene expression is greatly important for the study of NRG/ErbB4 signaling. Here we determined an approximate proportion of 85% glutamatergic neurons and 15% GABAergic interneurons in hippocampal cultures, prepared from C57BL/6 mice in embryonic (E) day 18, similar to the proportion reported in vivo and in vitro. We also determined the expression of Nrg1 type III, Nrg2 and ErbB4 genes by the day in vitro (DIV) 7. Nrg1 type III had higher expression levels than Nrg2, similar to the reported in the hippocampus in vivo. Together, these results suggest that these cultures can be used as a valid model for the study of NRGs signaling in vitro.]]></p></abstract>
<kwd-group>
<kwd lng="es"><![CDATA[Neuregulinas]]></kwd>
<kwd lng="es"><![CDATA[ErbB4]]></kwd>
<kwd lng="es"><![CDATA[neuronas primarias de hipocampo]]></kwd>
<kwd lng="es"><![CDATA[esquizofrenia]]></kwd>
<kwd lng="en"><![CDATA[Neuregulins]]></kwd>
<kwd lng="en"><![CDATA[ErbB4]]></kwd>
<kwd lng="en"><![CDATA[hippocampal neurons]]></kwd>
<kwd lng="en"><![CDATA[schizophrenia]]></kwd>
</kwd-group>
</article-meta>
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